Description
Ischaemia/reperfusion damage promotes pro-arrhythmogenic ventricular remodelling. The effects of histone deacetylase (DHA) enzyme inhibition on this process and its impact on electrophysiological properties and arrhythmogenesis will be discussed.
Keywords
ischaemia, reperfusion, cardiac remodelling, fibrosis, electrophysiology, arrhythmogenesis, DAH
Manager UV
- Chorro Gasco, Francisco Javier
- PDI-Emerit/a Universitat
Frascati classification
